By American Academy of Ophthalmology, Ramana S. Moorthy MD

ISBN-10: 161525563X

ISBN-13: 9781615255634

Starts with an outline of immune-mediated eye disorder, summarizing simple immunologic suggestions, ocular immune responses and precise issues in ocular immunology. Discusses the scientific method of uveitis and studies noninfectious (autoimmune) and infectious kinds of uveitis, with an elevated part on viral uveitis and new fabric on infectious and noninfectious scleritis.

Enhanced detection of infectious brokers by means of immunologic and genetic tools and biologic therapeutics are unique. additionally covers endophthalmitis, masquerade syndromes, issues of uveitis and ocular points of AIDS.

Upon final touch of part nine, readers will be capable to:

Identify basic and particular pathophysiologic tactics that have an effect on the constitution and serve as of the uvea, lens, intraocular cavities, retina, and different tissues in acute and protracted intraocular inflammation
Differentiate and determine infectious and noninfectious uveitic entities
Choose acceptable exam concepts and proper ancillary stories in line with even if an infectious or noninfectious reason is suspected

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Additional resources for 2014-2015 Basic and Clinical Science Course (BCSC): Section 9: Intraocular Inflammation and Uvetis

Example text

Walport MT. Complement. Second of two parts. N Engl JMed. 2001 ;344(15):1140-1144. Fibrin and other plasma factors Fibrin is the fin al deposition product of another important plasma-derived enzyme system, and its deposition during inflammation promotes hemostasis, fibrosis, angiogenesis, and leukocyte adhesion. Fibrin is released from its circulating zymogen precursor,fibrinogen, upon cleavage by thrombin. In situ polymerization of smaller units gives rise to the characteristic fibrin plugs or clots.

The contribution of histamine to intraocular inflammation remains subject to debate. Lipid Mediators Two groups of lipid molecules synthesized by stimulated cells act as powerful mediators and regulators of inflammatory responses: the arachidonic acid (AA) metabolites, or eicosanoids, and the acetylated triglycerides, usually called platelet-activating factors. Both groups of molecules may be rapidly generated from the same lysophospholipid precursors by the enzymatic action of cellular phospholipases such as phospholipase A2 (Fig 1-4).

Subsets of effector T lymphocytes can be divided into 2 main types by functional differences in experimental assays or by differences in cell surface molecules (Fig 2-5). Delayed hypersensitivity (DH) T lymphocytes usually express CD4 (and so recognize antigen with MHC class II) and release IFN-y and TNF-~. _ r@ Plasma cell Figure 2-5 Schematic representation of effector mechanisms during adaptive immunity. Not only is the immune response initiated within the tissue site, but ultimately the immune response arc is completed when effectors encounter antigen within the tissue after release into the circulation from the lymph node.

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2014-2015 Basic and Clinical Science Course (BCSC): Section 9: Intraocular Inflammation and Uvetis by American Academy of Ophthalmology, Ramana S. Moorthy MD


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